, Volume 65, Issue 1, pp 129–135

Trevor G. Marshall Trudy J. Rumann Heil


Studies in mice have shown that environmental electromagnetic waves tend to suppress the murine immune system with a potency similar to NSAIDs, yet the nature of any Electrosmog effects upon humans remains controversial. Previously, we reported how the human Vitamin-D receptor (VDR) and its ligand, 1,25-dihydroxyvitamin-D (1,25-D), are associated with many chronic inflammatory and autoimmune diseases. We have shown how olmesartan, a drug marketed for mild hypertension, acts as a high-affinity partial agonist for the VDR, and that it seems to reverse disease activity resulting from VDR dysfunction. We here report that structural instability of the activated VDR becomes apparent when observing hydrogen bond behavior with molecular dynamics, revealing that the VDR pathway exhibits a susceptibility to Electrosmog. Further, we note that characteristic modes of instability lie in the microwave frequency range, which is currently populated by cellphone and WiFi communication signals, and that the susceptibility is ligand dependent. A case series of 64 patient-reported outcomes subsequent to use of a silver-threaded cap designed to protect the brain and brain stem from microwave Electrosmog resulted in 90 % reporting “definite” or “strong” changes in their disease symptoms. This is much higher than the 3–5 % rate reported for electromagnetic hypersensitivity in a healthy population and suggests that effective control of environmental Electrosmog immunomodulation may soon become necessary for successful therapy of autoimmune disease.



PPPM:  predictive, preventive, and personalised medicine

NSAIDs: anti-inflammatory drugs


A photograph of a sleeping cap sewn from the microwave shielding fabric:



Immunopathology from Electrosmog

When the Electrosmog in a patient’s environment is reduced, the immune system tends to become more active. This may result in immunopathology. Indeed, some patients have reported a surge in disease symptoms, occasionally an intolerable surge, after WiFi routers and cell phones have been switched off in their homes. Others have reported that travel to a very quiet area, such as a remote canyon, caused a surge in their immune symptoms.

While further research is needed to clarify these reactions, autoimmune patients seem predisposed to Electrosmog hypersensitivity at levels currently existing in typical home and work environments, and this factor may be affecting their therapeutic response.

Finally, we need to plan how to handle subjects whose symptoms become untenable (due to immunopathology) during acclimatization to an Electrosmog-quiet environment, or during immune washout. We cannot ignore the increasing body of evidence showing electromagnetic effects on the immune system. The “controversial” nature of electromagnetic hypersensitivity will not diminish until we grasp the complexity of the task we face in defining exactly how electromagnetic waves interact with human biology.

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